Chintha Chandran*, Dhanya Dharman and Shaiju S. Dharan


INTRODUCTION The first study of the extrapyramidal side effects (EPS) of the antipsychotic chlorpromazine found that about 40% of these patients exhibited parkinsonism,[7] and several subsequent epidemiological studies found that DIP is the second most common etiology of parkinsonism. Drug-induced movement disorders include drug-induced parkinsonism (DIP), tardive dyskinesia (TD), tardive dystonia, akathisia, myoclonus, and tremor. Among these, DIP is the most common movement disorder induced by drugs that affect dopamine receptors.[1,3] Since the clinical manifestations of DIP are very similar to those of Parkinson's disease (PD), patients with DIP are frequently misdiagnosed as having PD.[1,4] These patients are often prescribed antiparkinsonian drugs unnecessarily for long periods of time, despite recovery being possible simply by discontinuing the offending drugs. Dopamine transporter (DAT) imaging may be used in the differential diagnosis of various etiologies of parkinsonism, including DIP.[5,6] The aim of this review was to provide clinicians with updated information about the clinical characteristics and DAT imaging findings of patients with DIP, and about the correct treatment for DIP.

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